The international team focused on the role played by the absence of a small fragment of chromosome 16 in the onset of a severe form of obesity. This anomaly more accurately called “microdeletion” (loss of a fraction of chromosome) causes the deletion of thirty different genes in this region. Researchers have discovered that this microdeletion, absent in most individuals, is quite common among the obese. They believe that the risk of developing severe obesity is greater than 5.000% for individuals carrying a deletion of chromosome 16. “If the identity of the genes responsible for obesity remains to be determined, it is likely that these genes play a role in brain development,” they note, because this chromosomal region is known to be linked to development of schizophrenia and autism, two diseases often complicated by obesity. These results also confirm the link between obesity and neuropsychiatric diseases.

But “obesity is multifactorial origin,” say the authors of that work. “It involves several genes, becoming better known, and environmental factors. Even if the increase in the number of overweight people has societal causes are easily identified (sedentary, food …), heredity plays a crucial role in determining the weight and the onset of obesity. Recent studies have revealed the involvement of thirty genes that increase the risk of obesity so low or moderate (increase of only 10 to 50% per gene).

Possibility Related Posts:

• The basics of a healthy and balanced diet
  Recognize and understand their mistakes food. o Situation 1: no breakfast. o Scenario 2: dinner too rich in calories. o Situation 3: skipping meals voluntarily or involuntarily. Situation 1: n...
• Venous Insufficiency
  Some 55% of women and 30% of men complain of heaviness in the legs late in the day. If we add hemorrhoids and pelvic pain in women, one realizes that the venous insufficiency a big part of us. Heav...